Know Your Number
Smoking · vaping · drinking  ·  the actual numbers

Know your
number.

No scare tactics, no lecture. Move the sliders, get a straight-up score for what your habits actually add up to, then dare a mate to check theirs.

The plot twist most people miss: cutting back doesn't cut your risk by the same amount. Doing it half the year isn't "half the risk" of year-round, the gap is way smaller than that. And mixing smoking or vaping with drinking doesn't just add the two risks together, it multiplies them.

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About EVALI: a 2019–2020 outbreak of serious vaping-linked lung injury (2,807 hospitalizations, 68 deaths, per the CDC) was traced almost entirely to vitamin E acetate in illicit THC vape carts, not regulated nicotine e-liquid. Real risk, tied to black-market products specifically, not something that scales with the sliders below.
Tobacco / Vaping

02040
1612
Alcohol
0510

1 standard drink = 14g pure alcohol (beer, wine, or a shot of spirits. The type doesn't matter much; the ethanol does).

1612
Duration
12550
Lifetime cigarettes0
Lifetime drinks0
Someone dared you to check your number.
Their number
--
Yours
--
--
--
/ 100
Your number
Crunching it...
Higher is better. 100 means you're barely moving the needle. The lower it drops, the more your habits are stacking up.
Biggest single risk: --
Biggest risk
--
--
Stacked vs added
--
what combining actually does
vs year-round
--
of the full-time risk
Lifetime tally
--
on this pattern
Lung cancer
1.0×
risk vs. someone who does neither

You

Year-round

Neither

1.0×
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Synergy: Lung

How your combined risk compares to simply adding the two separate risks together.

Combined risk by cancer type

Sorted highest to lowest, at your current settings.

Risk over time

Solid: your pattern. Dashed: same daily amounts, all year.

Your pattern
Both, year-round

Stuff worth knowing

Short, real, and honestly kind of wild.
01
20 min

That's roughly how long after your last cigarette your heart rate and blood pressure start dropping back toward normal. Your body doesn't wait around to start fixing things.

US Surgeon General
02
10 sec

Nicotine hits your brain in about ten seconds, faster than a shot through an IV. That speed is exactly why it grabs hold so hard and won't let go.

National Institute on Drug Abuse
03
×35

Heavy smoking and heavy drinking together push mouth and throat cancer risk to around 35 times someone who does neither. They don't add up, they multiply.

Jun et al., J Korean Med Sci 2024
04
~90%

Quit before about 40 and you dodge roughly 90% of the long-term risk of dying early from smoking. When you stop matters way more than most people think.

Jha et al., New England Journal of Medicine 2013
05
Zero

There's no amount of alcohol that's been shown to be safe when it comes to cancer risk. Even light, regular drinking nudges the odds up a little.

WHO 2023 · US Surgeon General 2025
06
Not vapor

Vape clouds aren't "just water." The aerosol carries ultrafine particles and trace metals that ride deep into your lungs, which is why the sliders here aren't zero.

National Academies of Sciences, 2018

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What this tool will never do

No tobacco, vape, or alcohol ads. Ever.

No selling your inputs. No targeted ads based on what you tell it. Display ads here are brand-safe, non-endemic, and category-blocked from alcohol and age-restricted products, configured at the ad-network level, not just promised in copy.

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How this is calculated, and where the numbers come from
The vaping model, specifically

Vaping is modeled with its own dedicated curves, not just a scaled-down cigarette curve, built from two separate evidence bases:

  • Cardiovascular risk is calibrated to pooled population data, not biomarkers. The curve is anchored to recent meta-analyses rather than any single study: a 2024–2025 network meta-analysis of 11 adult population studies found a pooled composite cardiovascular disease odds ratio of about 1.31 for e-cigarette use, and separate pooled estimates put the myocardial-infarction odds ratio at roughly 1.24–1.53 after adjusting for cigarette smoking. This tool sets every-day vaping heart-disease risk at about 1.5× and scales it down for less-frequent use. An earlier, widely-cited 2019 cross-sectional analysis of the PATH study reported higher figures (MI odds ratios of 1.99 some-day and 2.25 every-day), but that design is prone to reverse causation and sits above the pooled evidence, so it is treated here as an upper reference, not the anchor. Stroke is modeled at half the heart-disease excess (about 1.25× for every-day use), consistent with the one dataset examining vaping and stroke specifically finding a weaker, non-significant association (adjusted OR 1.13).
  • Cancer risk has no human incidence data yet: e-cigarettes haven't existed long enough for the decades-long latency most cancers require. This tool projects a cancer contribution from biomarker studies instead: NNAL (a tobacco-specific carcinogen metabolite) runs about 85–98% lower in exclusive e-cigarette users than in cigarette smokers across several independent studies. This tool scales vaping's contribution to roughly 10% of an equivalent cigarette's cancer-risk curve: a projection from exposure data, not a validated cancer-risk finding, and labeled as one throughout.
Why this varies so much by device: carbonyl/aldehyde output in e-cigarette aerosol has been measured anywhere from ~100–2,800× lower than cigarette smoke under normal use, to 5–15× higher under high-voltage "dry puff" conditions. This tool models a single central estimate; real risk for any individual device could reasonably fall outside it in either direction.

The often-repeated "e-cigarettes are 95% less harmful than smoking" figure traces to a 2014 UK expert-panel opinion exercise (not data-driven), popularized by a 2015 Public Health England report, and criticized in The Lancet for methodological weakness and conflicts of interest. This tool does not use that figure as a modeling input.

Why smoking/vaping + drinking multiplies risk instead of adding it

For cancers of the upper aerodigestive tract (mouth, throat, voice box, esophagus), alcohol acts partly as a solvent: it helps dissolve carcinogens and makes it easier for them to penetrate the cells lining the mouth and throat. The U.S. National Cancer Institute describes the combined harm at these sites as multiplicative. A 2024 meta-analysis (Jun et al., J Korean Med Sci) measured this directly for cigarettes and alcohol: heavy alcohol combined with heavy smoking produced roughly 35–39× the risk of neither. This tool's multiplicative model reproduces those figures within roughly 1–7%.

For vaping combined with alcohol, no equivalent study exists. This tool extends the same multiplicative mechanism to vaping as a projection, labeled as such wherever it appears.

Cancer types modeled, and which factor drives each one
Cancer typeSmoking ceilingAlcohol ceiling (6 drinks/day)Notes

"Smoking ceiling" is the relative risk approached at high cumulative pack-years for combustible cigarettes. For larynx, pharynx, oral cavity, and esophagus, the ceiling is scaled up from Gandini et al.'s pooled "current smoker" average by ~2.8×, the ratio separating lung cancer's population-average smoker risk from its long-term-heavy-smoker risk. That's what allows the multiplicative combination to match the JKMS heavy+heavy figures above.

Product multipliers
ProductCancer multiplierCardiovascular model
Regular / light / menthol cigarettes1.0×Standard cigarette dose-response curve
IQOS / heated tobacco0.85×Standard cigarette curve, scaled 0.85× (FDA: exposure-reduction claim allowed, disease-risk claim barred)
Vaping (e-cigarettes)~0.10× (projected)Dedicated curve anchored to pooled meta-analytic e-cig CVD odds ratios (~1.3-1.5)
Why heart disease and stroke don't include an alcohol effect

Alcohol's relationship to cardiovascular disease is genuinely contested science: some studies suggest lower risk at moderate intake, and more recent reviews dispute this. Rather than take a side, this tool models heart disease and stroke from tobacco/vaping only.

Important limitations
  • No study has tracked people who use these substances heavily for part of the year and stop, year after year. This tool approximates that as an averaged dose spread across the year, which is more likely to understate than overstate real risk.
  • EVALI is a real, serious, well-documented acute risk but isn't a graded dose-response relationship, so it's presented separately.
  • This is an educational model built from population averages, not a diagnostic or personalized medical risk assessment.
Sources referenced

National Academies of Sciences, Engineering, and Medicine, Public Health Consequences of E-Cigarettes, 2018 · Bhatta & Glantz, J Am Heart Assoc 2019 · e-cigarette CVD network meta-analysis, 2025 · Plurphanswat, Selya & Rodu, Cureus 2024 · CDC EVALI outbreak reports, 2019–2020 · NNAL biomarker studies, PATH / Cancer Epidemiology Biomarkers & Prevention 2025 · Public Health England 2015 review & The Lancet's critique · Jun S et al., J Korean Med Sci 2024;39(22):e185 · Gandini S et al., Int J Cancer 2008 · Cumberbatch MG et al., Eur Urol 2016 · U.S. Surgeon General's Advisory, Alcohol and Cancer Risk, 2025 · National Cancer Institute Alcohol and Cancer Risk Fact Sheet · Bagnardi V et al., Br J Cancer 2015 · Peters SAE et al., BMJ 2018 · NCI Smoking and Tobacco Control Monograph 13 · FDA Modified Risk Tobacco Product orders for IQOS.